Social Networking Essay Example for Free

Social Networking Essay In this report I will be outlining what Social Networking Sites are. A growing number of different Social Network Sites allows people to reunite and communicate without the limitations of distance or time. Enabling people to have regular interaction and keep up to date with the everyday life of others. So how do these sites work and why are they so popular? Associates may include close friends, family, distant relatives, old school friends, previous work colleagues or just shared interests. Whilst you will have frequent face to face contact with many of these people, others you will not. It is thought that our interactions with others, enhances our individual well-being. Due to the mainstream popularity of Social Network Sites where many people engage, they are a useful platform for both businesses and academic learning, with the creation of forum groups, they provide and extension of class discussion. Communication pages are a great benefit for schools and colleges to update information for parents and students. Not forgetting the functional applications, designed to support education. Many sites introduced games and other â€Å"applications† to entertain users and combat the flatness for new users during the early stages and those with a low number â€Å"friends†. Facebook has a large number of Applications available. Most sites contain common aspects, including a personal â€Å"profile† to allow the user to identify themselves, possibly with a photograph and a short description. They can then link to other people they know within the site to create a list of â€Å"friends†. Additional information may also be invited, such as age, location, hobbies and interests. ‘Their network of connections is displayed as an integral piece of their self-presentation’ (boyd et. al 2007). Some sites such as Twitter allows users to change the appearance of their profile page, this site is very popular with celebrities. As always, there are negative aspects to using online social network sites and the growing concerns surrounds the privacy methods in place. It is imperative that these sites are responsible with the control and use of data held for their users. Not only for the risk of data theft, but companies have been known to pass personal details on to third parties and used for unsolicited emails, also known as spamming. In addition to this, data mining software is repeatedly used to spy on regularly visited web pages and purchases to target the user to a more precise market. It is reported that many users have experienced cyber-bullying, which can have a major impact on personal health and well-being, causing anxiety and fear. Trolling is another increasing menace, where a user tries to impersonate another user, by setting up an account in another persons’ name using their pictures, intentionally causing upset by being offensive to others. Other risks include grooming, where sex pests try to build a trusting relationship online, with the aim to eventually meet. However, sites such as MySpace and Netlog are pro-active with the increasing number of sexual predators active on their site and often liaise with law enforcement to monitor and intercept such activities. Therefore, it is clear why many people prefer to completely avoid them. Launched in 1997, SixDegrees was the first Social Network Site. It had the same characteristics that we see today. Despite having millions of users, it did not develop into a worthwhile business. SixDegrees closed in 2000. The site creator deemed the site was ahead of it’s time. Today the top most popular Social Network Sites are: Facebook and Twitter. Strong contenders include MySpace, and LinkedIn. To summarise, I would describe Social Network Sites to be speed socialising, as we all have busy lives and may find it hard to make time for our close friends and family. This method enables people to socialise with a wider circle of friends you wouldn’t necessarily have time to socialise with. It is important to be aware of the personal details we are supplying about ourselves and the consequences it could have, it may seem obvious but it is essential that people should only share information that you are happy for others to know. But, if used with both common sense and caution the sites can be very positive and gratifying. [Word Count: 700] References DirectGov, 2012. Social Networking Service [online] Available at: http://www.direct.gov.uk/en/Parents/Yourchildshealthandsafety/Internetsafety/DG_182627 [Accessed 13 April 2012] Boyd, d. m. and Ellison, N. B. (2007) ‘Social network sites: definition, history, and scholarship’, in Donelan, H., Kear, K. and Ramage, M. (eds) Online Communication and Collaboration: A Reader, Abingdon, Routledge, pp. 261–281.

The Importance of Spontaneity in Times Square :: Free Essays Online

The Importance of Spontaneity in Times Square 1. The typical large, industrial city can only be described as constantly alive. Crowded streets, sidewalks, restaurants, shops, and apartments all contribute to this aspect of major cities of the world; the bustling and vibrating streets are only indicative of the people who continue to flock to cities for opportunity in participating in the success that has obviously implemented their growth. While city dwellers are attracted to the prospect of fortune and accomplishment, what they might not realize is that their interactions with one another actually create the lively atmosphere. In reference to this idea, one can examine the characteristics of Times Square in New York City. While some areas of the city are actually like small communities, such as apartment buildings and other living spaces which are clustered together, Times Square is more of a commercial space, therefore, a constant motion exists amongst the consumers and commuters that frequent the area. In spaces su ch as these, there is no structure of community because there are so many strangers pushed into a space and individuals usually do not create any sort of relationship with those that they encounter on a given day. However, is this constant bustle not responsible for so many people from around the world being attracted to Times Square? Because Times Square lacks the structure of community that one may find in rural areas or in the noncommercial areas of New York City, a certain spontaneity results that gives the space its own distinct personality and existence. 2. In her essay â€Å"Cities for Sale: Merchandising History at South Street Seaport†, M. Christine Boyer argues that the atmosphere of certain areas in New York City, such as Times Square, has become fake. Boyer compares these spaces to Disneyland in her essay because she firmly believes that many areas in the cities of today have been reconstructed to create a sense of fantasy and security: â€Å"At Disneyland, the American way of life is displayed as a universal sign of progress† (Boyer 200). People are attracted to this type of environment because it creates a dreamland where they can escape reality, and they exist in this dreamland through buying the products that the space has to offer. The Importance of Spontaneity in Times Square :: Free Essays Online The Importance of Spontaneity in Times Square 1. The typical large, industrial city can only be described as constantly alive. Crowded streets, sidewalks, restaurants, shops, and apartments all contribute to this aspect of major cities of the world; the bustling and vibrating streets are only indicative of the people who continue to flock to cities for opportunity in participating in the success that has obviously implemented their growth. While city dwellers are attracted to the prospect of fortune and accomplishment, what they might not realize is that their interactions with one another actually create the lively atmosphere. In reference to this idea, one can examine the characteristics of Times Square in New York City. While some areas of the city are actually like small communities, such as apartment buildings and other living spaces which are clustered together, Times Square is more of a commercial space, therefore, a constant motion exists amongst the consumers and commuters that frequent the area. In spaces su ch as these, there is no structure of community because there are so many strangers pushed into a space and individuals usually do not create any sort of relationship with those that they encounter on a given day. However, is this constant bustle not responsible for so many people from around the world being attracted to Times Square? Because Times Square lacks the structure of community that one may find in rural areas or in the noncommercial areas of New York City, a certain spontaneity results that gives the space its own distinct personality and existence. 2. In her essay â€Å"Cities for Sale: Merchandising History at South Street Seaport†, M. Christine Boyer argues that the atmosphere of certain areas in New York City, such as Times Square, has become fake. Boyer compares these spaces to Disneyland in her essay because she firmly believes that many areas in the cities of today have been reconstructed to create a sense of fantasy and security: â€Å"At Disneyland, the American way of life is displayed as a universal sign of progress† (Boyer 200). People are attracted to this type of environment because it creates a dreamland where they can escape reality, and they exist in this dreamland through buying the products that the space has to offer.

Endothelial tight junction proteins Essay

Endothelial tight junction proteins Introduction            The endothelium is situated at the inner side of all kinds of vessels and comprises of a monolayer of endothelial cells. Inter-endothelial junctions comprise junctional complexes, such as adherens junctions (AJ), tight junctions (TJ) and gap junctions (GJ) that play essential roles in tissue integrity, barrier function and intercellular communication respectively. These junctional complexes are related to those found at epithelial junctions with notable changes in terms of certain molecules and structure.            Endothelial junctional proteins play important roles in tissue integrity but also in vascular permeability, leukocyte extravasation and angiogenesis. Dormant endothelium may be exposed to stimuli provoking leukocyte extravasation at seditious sites and propagating angiogenesis. Both activities have an intense impact on endothelial cell-cell junctions.            Tight junctions aid the major functional objective of establishing a barrier inside the membrane, by controlling paracellular permeability and sustaining cell polarity. They achieve this by constricting apical or basolateral transmembrane diffusion of lipids and they have been suggested to contribute in regulating proliferation and differentiation of epithelial cells. However, the components that are involved and the signal routes concerned are unknown (Mitic & Anderson 1998).            Tight junctions are made up of integral membrane proteins claudins, occludin, tricellulin, junctional adhesion molecules (JAMs), including many peripheral membrane proteins such as the scaffold PDZ- domain proteins. This review will however, focus on ZO-1 and ZONAB. Histology of endothelia junctions            The junctional structures situated at the endothelial intercellular fissure are related to those located at the epithelium; however, their formation is more inconsistent and in most vascular beds their topology is less constrained than in epithelial cells. Adherens junctions, tight junctions and gap junctions are in most cases intermingled and create a complex zonular system with disparities in depth and thickness of the sub-membrane plate associated with the junctional structure (Franke et al. 1988; Rhodin 1974). In contrast to epithelial cells, GJs are often found close to the luminal surface. Hence, the term â€Å"Apical junction† used to jointly describe epithelial TJ and AJ may not be applied to the endothelium. The endothelium forms the vascular barrier with controlled permeability properties between the blood and the underlying tissues.            Tight junctions exhibit considerable inconsistency among different segments of the vascular tree (Franke et al. 1988). This disparity composes a major evidence of vascular bed differentiation of endothelial cells and has a strong impact on vascular permeability and leukocyte extravasation. Variations concern the complexity degree of the occluding strands as well as tight junction composition.            Large Artery endothelial cells, which are exposed to high flow rates, display a well-developed system of tight junctions. Within the microvasculature, tight junctions are less complex in capillaries than in arterioles, and even less in venules. It is important to mention that, post-capillary venules are the primary site of leukocyte extravasation, and accordingly, they display a high content of permeability mediator receptors, such as those for histamine, serotonin and bradykinin. On the other hand, blood brain barrier (BBB) and the blood retinal barrier (BRB) are predominantly rich in Tight Junctions and endothelial tight junctions have been principally studied in these sites.            Endothelial intercellular realms differ from those of epithelial cells by the absence of desmosomes (Franke et al. 1988). The transitional filaments, comprised in the endothelium by vimentin molecules, are poorly connected to cell-cell contacts. However, contrary to the situation in epithelia, the vimentin filaments may be associated to endothelial adherens junctions in junctional structures similar to desmosomes, called complexus adherens.            It must be emphasized that interendothelial junctions are vibrant structures, subjected to multiple regulations. Moreover, leukocytes extravasate majorly in postcapillary venules either through transcellular or paracellular methods. Extravasation via the intercellular junction is a rapid and controlled process, through which the leukocyte is squeezed in the fissure (diapedesis), followed by rapid junction reformation.            ZO-1 is a protein located on the cytoplasmic membrane plate of intercellular tight junctions and is engaged in transducing signals at cell-to-cell junctions. ZO-1 links tight junction transmembrane proteins to a cytoplasmic plaque and the actin-based cytoskeleton (Aijaz et al. 2006; Tsukita et al. 2001). In epithelial cells, ZO-1 interrelates with the transcription factor ZONAB to regulate cells proliferation in a cell density related manner (Balda & Matter 2000); however, the functions of ZO-1 and ZONAB in endothelial cells are still not clearly understood.            Unpublished work shows that downregulation of ZO-1 in endothelial cells stimulates redistribution of two transmembrane proteins; claudin-5 and JAM-A, and radical changes in the cytoskeleton affecting the localization of mechanosensor proteins and VE-cadherin role in the control of cell-cell tension.            These observations imply that one function of ZO-1 in endothelial cells is to coordinate components of the tight junction and associate them to the cortical cytoskeleton. However, it is unfamiliar whether the ZO-1 associated transcription factor ZONAB is linked to such ZO-1 effects.            Despite the fact that, ZO-1 explicitly associates with epithelial tight junctions (Stevenson et al. 1986), it has been observed that the protein appears in the nucleus in the process of proliferation (Gottardi et al. 1996). While the functional impact of the nuclear localization is currently not clear, studies reveal that these discrete subcellular distributions of ZO-1 are exquisitely sensitive to the state of cell-to-cell contact.            ZO-1 plays a major role of restraining ZONAB and regulates its accumulation in the nucleus through cytoplasmic sequestration. MDCK cells found in the epithelium exhibit two forms of this Y-box transcription factor (ZONAB) i.e. ZONAB -A and ZONAB -B which vary in a 68-amino acid supplement. Both categories of ZONAB bind to ZO-1 and link with intercellular junctions (Balda & Matter 2000).            ZONAB was initially designated in canine kidney epithelial cells (MDCK) and is a Y-box transcription factor. Y-box transcription factors are multipurpose control mechanisms of gene expression and studies suggest that they play a common role in enhancing proliferation (Bargou et al. 1997). ZONAB is one of the tight junction-associated dual localization protein: it localizes to junctions where it attaches to the SH3 surface of the adaptor protein ZO-1, and to the nucleus where it regulates transcription.            The distribution of ZONAB is controlled by the cell density as it localizes to both junctions and nuclei in low density, proliferating cells, and becomes constrained to the cytoplasm in high density cells (Balda & Matter, 2000). This distribution is also exhibited in its transcription activity, as ZONAB is transcriptionally vigorous in proliferating cells but inactive in non-proliferating cells. In the MDCK cells, ZONAB is necessary for normal rates of proliferation and controls G1/S phase transition (Balda et al. 2003).            ZONAB affects cell cycle development by two distinct processes: it controls the nuclear accumulation of CDK4 through a direct interaction and controls manifestation of genes encoding cell cycle regulators for example, PCNA and cyclin D1 (Balda et al. 2003; Sourisseau et al. 2006 ).            In 3D principles of MDCK cells, regular ZO-1 and ZONAB processes are necessary for epithelial cyst formation, implying that the Y-box transcription factor also controls epithelial differentiation (Sourisseau et al. 2006). Since ZO-1 and ZONAB can also relate with other types of intercellular junctions, for instance the gap junctions, in cells that lack tight junctions, it is possible that ZO-1 or ZONAB signaling is also of useful significance in other cell types other than epithelia (Ciolofan et al. 2006; Giepmans & Moolenaar 1998). Aims of the study            The aim of the study is to understand the functional consequences of downregulation of ZONAB in endothelial cells, and whether and how ZONAB cross-talks with other junctional components to regulate endothelial cell migration, proliferation and angiogenesis. Currently, we are looking at similarities and differences between the phenotype of downregulation of ZO-1 or ZONAB by RNA interference. Changes in expression and localization of a given protein are analysed using specific antibodies for immunoblots and immunofluorescence. Preliminary Results            It is observed that downregulation of ZO-1 or ZONAB resulted in similar redistribution of actin and vinculin from cell-cell junctions to stress fibers and focal adhesions, respectively. However, the localization of transmembrane proteins such as Claudin-5 and JAM-A is affected by downregulation of ZO-1 rather than by downregulation of ZONAB. The localization of the polarity protein PAR-3 is changed in both conditions.            Additionally, downregulation of ZONAB causes changes in ZO-1 by immunofluorescence that needs to be tested for expression by immunoblots. Next, we will characterize other transmembrane proteins (e.g. MD3 and claudin-1), polarity proteins (PKCzeta), Rho regulators and mechanotransducers such as PAK2, Zyxin and YAP.            ZONAB is a DNA and RNA binding factor that it is involved in transcription (e.g. cyclin D1 and PCNA) in the nucleus and translation (e.g. cell cycle inhibitor p21) in the cytosol. Thus, we are also trying to identify new genes regulated. We have identified that expression of fibronectin is regulated by ZONAB. We are evaluating whether the changes in protein expression of fibronectin are due to ZONAB role on transcription or translation, using actinomicin D to inhibit transcription or cyclohexidimide to inhibit translation. Additionally, we are validating new genes identified by cDNA array analysis of endothelial cells with downregulation of ZONAB.            The tight junction localizing protein ZO-1 symptomatically forms a continuous band around the apices of well-differentiated, confluent, polarized epithelial cells in culture. However, under nonconfluent conditions, endogenous ZO-1 can localize to the nucleus in addition to the border of cell-cell contact.            ZONAB manifestation tends to be high in proliferating but low in growth-impeded MDCK cells, implying that high manifestation levels might be a necessity for cell proliferation (Balda & Matter 2000).            ZONAB confines in the nucleus as well as tight junctions in proliferating cells, however, it is not noticeable in the nucleus of nonproliferating high density cells (Balda & Matter 2000), proposing that accumulation of ZONAB in the nucleus might be necessary for efficient proliferation.            ZO-1 quantities are low in proliferating cells and they rise with cell density, and overexpression of ZO-1 hinders accumulation of ZONAB in the nucleus (Balda & Matter 2000); hence, ZO-1 may control proliferation by inhibiting ZONAB from accumulating in the nucleus. Overexpression of ZO-1 in low density cells triggers a redistribution of ZONAB from the nucleus to the cytoplasm and reduced proliferation.            CDK4 is a major regulator of G1/s transition (Sherr 2000; Malumbres & Barbacid 2001). Thus, ZONAB could control proliferation by regulating the process or the localization of CDK4. Since ZONAB binds CDK4, the nuclear pools of the two proteins may diminish in a parallel manner.            Symplekin is combined with ZONAB in the nucleus; hence, it could be argued that Symplekin modulates the transcription activity of ZONAB. Increased expression of Symplekin results in stimulation of the transcriptional suppressor ZONAB. However, it is also noted that Symplekin is absent in endothelial cells (Keon et al. 1996).            ZONAB controls cell cycle entry. ZO-1 overexpression results in a reduction in DNA synthesis, implying that entry into S-phase was distressed.            These experiments will allow understanding the role of ZO-1 and ZONAB in endothelial cells. Depending on the results, we plan to test how these two proteins are involved in endothelial stress conditions such as shear stress and high glucose. Conclusion            The collaboration of ZO-1 with tight junctions can only be significant for the stabilization of ZO-1, as opposed to attaching ZO-1 to the plasma membrane so as to constrain nuclear accumulation of related proteins. This is supported by the opinion that a truncated protein comprising only the HA-tagged SH3 domain accumulated in the Cytosol, but was adequate to decrease proliferation and nuclear accumulation of ZONAB (unpublished data).            ZONAB and ZO-1 control proliferation and the ultimate cell density of MDCK cells. Explanations that ZO-1 accumulates with increasing cell density, and overexpression of ZO-1 in transfected cells lowers the final density proposes a pattern in which ZO-1 serves as a measure for cell density whereby, on reaching the threshold level, provokes growth impediment by cytoplasmic sequestration of ZONAB and the related cell cycle kinase CDK4. It will be essential to control how the ZO-1 or ZONAB pathway associates with the other signaling methods that affect proliferation.            Vascular endothelial stress induces dysfunctions that have been implicated in many diseases such as diabetes and diabetic retinopathy. Therefore, characterization of the role of tight junction molecules in different endothelial cell behavior and functions will help us to understand the molecular mechanisms of disease pathogenesis and these findings may be implicated in prognosis and possibly to develop new treatment strategies. References Balda, MS and Matter, K 2000. The tight junction protein ZO-1 and an interacting transcription factor regulate ErbB-2 expression. EMBO J. 19, pp 2024-2033. Balda MS, Garrett MD and Matter K, 2003. The ZO-1 associated Y-box factor ZONAB regulates epithelial cell proliferation and cell density. J. Cell Biol. 160, pp 423-432. Bargou RC, K Jurchott, C Wagener, S Bergmann, S metzner, K Bommert, MY Mapara, KJ Winzer. M Dietel, B Dorken, and HD Royer, 1997. Nuclear localization and increased levels of transcription factor YB-1 in primary human breast cancers are associated with intrinsic MDR1 gene expression. Nat. Med. 3: pp 447-450. Ciolofan C, Li XB, Olson C, Kamasawa N, Gebhardt BR, Yasumura T, Morita M, Rash JE and Nagy JI, 2006. Association of connexin36 and Zonula occludens-1 with zonula occludens-2 and the transcription factor zonula occludens-1 associated nucleic acid-binding protein at neuronal gap junctions in rodent retina. Neuroscience 140: pp 433-451. Franke WW, P Cowin, C Grund, C Kuhn, HP Kapprell, 1998, The Endothelial Junction: the plaque and its component., in: N. Simionescu, M Simionescu (Eds.), Endothelial cell biology in health and diseases, Plenum publishing corporation, New York. pp 147-166. Giepmans BN and Moolenaar WH, 1998. The gap junction protein connexin43 interacts with the second PDZ domain of the zonal occludens-1 protein. Curr. Biol. 8. Pp 931-934. Gottardi CJ, M Arpin, AS Fanning and D Louvard, 1996. The junction-associated protein, zonular occludens-1, localizes to the nucleus before the maturation and during the remodeling of cell-cell contacts. Proc. Natl. Acad. Sci. USA. 93: pp 10779-10784. Keon BH, S Schafer, C Kuhn, C Grund, WW Franke, Symplekin, a novel type of tight junction plaque protein, J Cell Biol. 134 (1996) 1003-1018.Malumbres M and M Barbacid, 2001. To cycle or not to cycle: a critical decision in cancer. Nat. Rev. Cancer. 1: pp 222-231. Mitic LL and JM Anderson, 1998. Molecular architecture of tight junctions. Annu. Rev. Physiol. 60: pp 121-142. Rhodin, JAG 1974, Histology, Oxford University Press, New York. Sherr, CJ 2000. The Pezcoller lecture: cancer cell cycles revisited. Cancer res. 60: pp 3689-3695. Sourisseau T, Georgiadis A, Tsapara A, Ali RR, Pestell RG, Matter K and Balda MS, 2006. Regulation of PCNA and cyclin D1 expression and epithelial morphogenesis by the ZO-1 regulated transcription factor ZONAB/DbpA. Mol. Cell. Biol. 26, pp 2387-2398.Stevenson, BR, JD Siliciano, MS Mooseker, and DA Goodenough, 1986. Identification of ZO-1: a high molecular weight polypeptide associated with the tight junction (zonula occludens) in a variety of epithelia. J. Cell Biol. 103: pp 755-766. Source document

Enron - 750 Words

Enron: Ethics and Auditing Gone Wrong Enron was once a promising company headed toward greatness but all of this was just for show and thus not long enough; it was discovered that one of the world’s most admired companies was just faking all their records taking down a lot of investors of their company to bankruptcy as well as their employees. The Enron scandal has paved the way not only to America’s consciousness on risks involved on how corporations work, but how stakeholders can be victimized by fraud if corporations are not properly audited and regulated. It also opened consciousness of the whole world as well. In this paper, we will take a look on how proper implementation of business ethics and auditing can help avoid tragedies†¦show more content†¦During this time, Enron was ranked as the most innovative American company in Fortune magazine’s survey of Most Admired Companies (2). While the stocks are rising, the executives cash-in their option in a process known as â€Å"pump and dump† (1). Heavy PR campaigns are also made to project profitable and stable business operations despite the fact that the actual performance of the business worldwide is crumbling. Fall of Enron To enforce his directives, Skilling hired people known as the â€Å"guys with spikes† within Enron. Unfortunately these leaders had personal issues as well coming in the mix. J. Clifford Baxter who is assigned as an executive is manic-depressive; Lou Pai, CEO of Enron Energy Services on the other hand notoriously uses shareholder’s money visiting strip clubs. Lou Pai resigns making $250MM after he sold his stocks despite the fact that the divisions he led lost $1 billion (the loss is covered by Enron). Enron’s competitive advantage, as well as its huge profit margins, had begun to erode by the end of 2000. Energy prices began to fall in the first quarter of 2001 and the world economy headed into a recession, thus dampening energy market unpredictability and reducing the opportunity for the large, rapid trading gains that had formerly made Enron so profitable. (1). Enron’s foundations were developing cracks and Skilling’s house of paper built on the stilts of trust had started to crumble and eventually wasShow MoreRelatedEnron Of Enron And Enron1209 Words   |  5 PagesEnron Cooperation, is a company that was based in Houston Texas and was an energy company. This company filed bankruptcy in 2001 leaving a lot of its employees that had no knowledge about what was going on jobless and the company investors losing a lot of money. This was one of biggest companies in the united states, it had a lot of assets all over the country and was operating on a lot of prof it that nobody knew how and why. 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